Stroke subtype-dependent synapse elimination by reactive gliosis in mice

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Astrocytes mediate synapse elimination through MEGF10 and MERTK pathways. - Abstract - Europe PMC

Observing Extracellular Vesicles Originating from Endothelial Cells in Vivo Demonstrates Improved Astrocyte Function Following Ischemic Stroke via Aggregation-Induced Emission Luminogens

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Stress induces behavioral abnormalities by increasing expression of phagocytic receptor MERTK in astrocytes to promote synapse phagocytosis - ScienceDirect

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Stroke subtype-dependent synapse elimination by reactive gliosis in mice

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Lesion environments direct transplanted neural progenitors towards a wound repair astroglial phenotype in mice