Herpes simplex virus enhances chemokine function through modulation of receptor trafficking and oligomerization

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Last updated 16 julho 2024
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Frontiers Modulation of cellular signaling by herpesvirus-encoded G protein-coupled receptors
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Viral G Protein–Coupled Receptors: Attractive Targets for Herpesvirus-Associated Diseases
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
HSV-2 glycoprotein J promotes viral protein expression and virus spread - ScienceDirect
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Full article: Pathogenesis and virulence of herpes simplex virus
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Remodeling our concept of chemokine receptor function: From monomers to oligomers - Martínez‐Muñoz - 2018 - Journal of Leukocyte Biology - Wiley Online Library
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Innate metabolic responses against viral infections
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Viral G Protein–Coupled Receptors: Attractive Targets for Herpesvirus-Associated Diseases
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Herpes Simplex Virus 2 Counteracts Neurite Outgrowth Repulsion during Infection in a Nerve Growth Factor-Dependent Manner
Herpes simplex virus enhances chemokine function through modulation of  receptor trafficking and oligomerization
Full article: Pathogenesis and virulence of herpes simplex virus

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