Histone methyltransferases Setd1b increases H3K4me3 level to
Por um escritor misterioso
Last updated 10 março 2025


SETD1B controls cognitive function via cell type specific regulation of neuronal identity genes

The H3K4 methyltransferase Setd1b is essential for hematopoietic stem and progenitor cell homeostasis in mice

Histone Lysine Methylation Dynamics: Establishment, Regulation, and Biological Impact - ScienceDirect

Emerging roles of H3K9me3, SETDB1 and SETDB2 in therapy-induced cellular reprogramming, Clinical Epigenetics

Histone methyltransferase and histone methylation in inflammatory T-cell responses

The HIF complex recruits the histone methyltransferase SET1B to activate specific hypoxia-inducible genes

Frontiers SETD1A Mediated H3K4 Methylation and Its Role in Neurodevelopmental and Neuropsychiatric Disorders

Postnatal SETD1B is essential for learning and the regulation of neuronal plasticity genes

Histone methyltransferase and histone methylation in inflammatory T-cell responses

Frontiers Function and Regulation of Histone H3 Lysine-4 Methylation During Oocyte Meiosis and Maternal-to-Zygotic Transition

Dynamic mRNA degradome analyses indicate a role of histone H3K4 trimethylation in association with meiosis-coupled mRNA decay in oocyte aging

Roles and regulation of histone methylation in animal development

Expression of the histone lysine methyltransferases SETD1B, SETDB1, SETD2, and CFP1 exhibits significant changes in the oocytes and granulosa cells of aged mouse ovaries

SETD1B controls cognitive function via cell type specific regulation of neuronal identity genes
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