The ALS Association - Knock ALS Out of the Park

TDP-43 loss and ALS-risk SNPs drive mis-splicing and depletion of UNC13A

The ALS Association - Knock ALS Out of the Park

Genetics of amyotrophic lateral sclerosis: seeking therapeutic targets in the era of gene therapy

Targeting mitochondrial Ca2+ uptake for the treatment of amyotrophic lateral sclerosis - Zhong - The Journal of Physiology - Wiley Online Library

Cholesterol dyshomeostasis in amyotrophic lateral sclerosis: cause, consequence, or epiphenomenon? - Hartmann - 2022 - The FEBS Journal - Wiley Online Library

Don't look away., ALS can knock me down as many times as it wants. But this stubborn Irish SOB will never stop getting up., By The ALS Association

Selective neuronal degeneration in MATR3 S85C knock-in mouse model of early-stage ALS

Genes, Free Full-Text

Integrative genetic analysis illuminates ALS heritability and identifies risk genes

The ALS Association - Knock ALS Out of the Park

Postmortem Cortex Samples Identify Distinct Molecular Subtypes of ALS: Retrotransposon Activation, Oxidative Stress, and Activated Glia - ScienceDirect

Loss of the metabolism and sleep regulating neuronal populations expressing orexin and oxytocin in the hypothalamus in amyotrophic lateral sclerosis - Gabery - 2021 - Neuropathology and Applied Neurobiology - Wiley Online Library

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The ALS Association - Knock ALS Out of the Park